GDNF Antibody

ArtNr	OABB01534
Hersteller	AVIVA Systems Biology
Menge	100 ug/vial
Kategorie	Neuroscience Neural Signaling Antibodies Immunohistochemistry Western Blot ELISA Immunocytochemistry Primary Antibodies Immunostaining
Typ	Antibody Polyclonal
Format	Liquid
Applikationen	WB, ELISA
Specific against	Human (Homo sapiens)
Host	Rabbit
Isotype	IgG
Konjugat/Tag	Unconjugated
ECLASS 10.1	32160702
ECLASS 11.0	32160702
UNSPSC	12352203
Alias	CASP-2;caspase 2 apoptosis-related cysteine peptidase;caspase-2;ICH1;NEDD2;NEDD-2;neural precursor cell expressed developmentally down-regulated protein 2;PPP1R57;protease ICH-1;protein phosphatase 1,regulatory subunit 57.
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Manufacturer - Type	Polyclonal Antibody
Manufacturer - Category	Root Catalog/Products/Polyclonal Antibodies, Root Catalog/Products/Primary Antibodies
Gene symbol	CASP2
Gene Fullname	caspase 2
Product format	Liquid
Reconstitution and storage	-20°C or -80°C
Description of target	Glial cell line-derived neurotrophic factor(GDNF) is a glycosylated, disulfide-bonded homodimer that is a distantly related member of the transforming growth factor-beta superfamily. GDNF is also a potent neurotrophic factor that promotes the survival of dopaminergic neurones in cultures including embryonic neuronal cultures. In addition to its potential role in the differentiation and survival of central nervous system neurons, it has profound effects on kidney organogenesis and the development of the peripheral nervous system. GDNF may have utility in the treatment of Parkinson's disease, which is marked by progressive degeneration of midbrain dopaminergic neurons.
Protein name	Caspase-2
Clonality	Recombinant Monoclonal
Purification	Affinity Purified
Immunogen	A synthesized peptide derived from human Caspase-2
Dilution	Involved in the activation cascade of caspases responsible for apoptosis execution. Might function by either activating some proteins required for cell death or inactivating proteins necessary for cell survival.

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OABB01534-datasheet.pdf

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Menge: 100 ug/vial

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Lieferung vsl. bis 27.11.2025

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